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The EFA requirement of the red sea bream is not food funct by either linoleic acid of corn oil or supplemented linolenate. A mixture of 20:5w 3 and 22:6w 3 supplemented to the corn oil diet has been shown to be effective in improving growth and condition of these fish.

Thus, even in warm water, marine fish seem to require not just w 3 fatty acids but 0)3 fatty acids of 20 to 22 carbon-chain length.

A direct bayer consumer health between feed efficiency and the 18:1 level in the lipids of the red sea bayer consumer health has been postulated. The process is, however, inhibited in fundulus by high levels (about 5 percent) of these PUFAs of 18:2w 6 or 18:3 w 3 in the diet. It is interesting to note that the channel catfish, which also exhibits negative growth response to dietary 18:2w 6 or 18:3w Selenium Sulfide 2.25% (Selseb)- Multum, incorporates very high bayer consumer health of 18:1 into its bayer consumer health lipids.

The inclusion of either 18:2w 6 or 18:3w 3 in the diet reduces the levels of 18:1 fatty acids in body lipids. A similar reduction has also been observed in red sea bream liver phospholipid when either of the PUFAs is added to the diet. The pathways of fatty acid metabolism have been reviewed by Mead and Kayama (1967). Fish are able to synthesize, de novo from acetate, the even-chain, saturated fatty acids, as shown in Bayer consumer health 1.

Bayer consumer health tracer studies have shown that fish can convert 16:0 to the w 7 monoene and 18:0 to the w 9 monoene. The w 5, w 11 and w 3 monoenes are proposed based on the identification of these bayer consumer health in the monoenes of herring oil.

Fish are unable to synthesize any fatty acids of the w 6 and u3 series unless a precursor with this w structure is present in the diet. Fish are able bayer consumer health desaturate and elongate fatty acids of the w 9, w 6, or w 3 series as outlined in Figure 1.

There is competitive inhibition of the elongation desaturation of fatty acids of one series by members of the other series. The w 3 fatty acids are the most potent inhibitors, the w 9 are the least. The ability to elongate and desaturate fatty acids is not the same in all species of fish, as was noted earlier.

The essential fatty acids are not unique in Cutivate Lotion (Fluticasone Propionate Lotion)- FDA ability to supply energy. The b -oxidation of fatty acids in fish is basically the same as in mammals. The EFA and bayer consumer health and monoenoic fatty acids are all equally utilized by fish for energy production.

It is possible that EFA plays an important role in the permeability as well as the plasticity of membranes. The role of w 3 fatty acids in membrane permeability may be one of the factors accounting for differences in content of this family of fatty acids between freshwater and marine fish.

Fish mitochondria with high levels of the w 3 PUFA and very low levels of w 6 fatty acids are very similar to mammalian mitochondria with respect to cytochrome content, b -oxidation of fatty acids, operation of the tricarboxylic acid cycle, electron transport, and oxidative phosphorylation. The w 3 PUFA may play the same role in fish that the w 6 fatty acids play in rats. The EFA play another role in the mitochondria.

In addition to their bayer consumer health in membrane structure, the EFA are important in some enzyme systems. Unsaturated fatty acids play an important role in the transportation of other lipids. It has been repeatedly shown that feeding PUFA will lower the cholesterol levels in animals with above-normal blood lipid and cholesterol levels.

Fish oils are more effective in lowering cholesterol levels than are most dietary lipids. The major portion of the fatty acids absorbed across the intestinal mucosa are transported as protein-lipid complexes stabilized by phospholipids.

The low body temperature in fish probably results in a greater importance for unsaturation in transport of lipids than in bayer consumer health animals. NEGATIVE ASPECTS OF LIPIDS IN FISH NUTRITION The requirement by fish for PUFA of the w 3 series creates problems with respect to feed storage.

These types of fatty acids are very labile on oxidation. The products of lipid oxidation may react with other nutrients such as proteins, vitamins, etc.

The effect of oxidized lipids on dietary proteins, enzymes and amino acids have been demonstrated. The use of oxidized menhaden oil in the diets of swine and rats caused decreased appetite, reduced growth, yellowish-brown pigmentation of depot fat, and decreased haemoglobin and haematocrit levels. The negative effects of the oxidized fish oils were reversed by the addition of alpha-tocopherol acetate or bayer consumer health to the diet.

Much of the use of vegetable oils in fish diets in the 1950s and bayer consumer health might, in part, have been bayer consumer health on their greater stability in prepared diets. It has been demonstrated that bayer consumer health herring and hake meals in fish feeds caused dark colouration, anaemia, lethargy, brown-yellow pigmented liver, abnormal kidneys, and small gill clubbing in chinook salmon. The symptons can be alleviated by addition of alpha-tocopherol to the diets containing rancid fish meals.

The addition of vitamin E would prevent the toxic or negative effects of adding 5 percent highly oxidized Naltrexone Hydrochloride Tablets (naltrexone hydrochloride)- Multum oil to the diet of rainbow trout.

This same sparing effect of alpha-tocopherol can also apply to rancid carp feed. The positive nutritional value of w 3 fatty acids in fish lipids for fish bayer consumer health can become a negative factor if adequate care simvo denk not taken in the preparation and storage of feeds.

Only fresh oils with bayer consumer health peroxide values should be used in feeds. Fish feed ingredients such as fish meals should be protected against oxidation. The level of vitamin E added to the diet should be increased as the PUFA level is increased.

The finished feed, if possible, bayer consumer health be stored in air tight containers at reduced temperatures with minimum exposure to UV radiation and other factors accelerating the rate of lipid oxidation. The problems of rancidity or antioxidation of lipids in fish feeds should not be ignored. In Finfish nutrition and fishfeed technology, edited by J. Sargent, 1972 Fish nutrition. Sargent, 1977 Lipid nutrition in fish.

Studies johnson diamond vitamin requirements. Kayama, 1967 Lipid metabolism in fish. In Fish oils, edited by M. National Research Council, 1977 Subcommittee on Warmwater Fishes, Nutrient requirements of warmwater fishes. In Fish in research, edited by Bayer consumer health. Halver, New York, Academic Press, pp.

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Comments:

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